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Klotho Protein

The Longevity Factor · February 17, 2026

Summary

Klotho is a protein that acts as a master regulator of aging. Named after the Greek goddess who spins the thread of life, it protects the brain, heart, and kidneys while suppressing inflammation and oxidative stress. People with higher Klotho levels live longer and stay cognitively sharper — and scientists are now racing to develop drugs that boost it.

1
Elementary School
Ages 8-10

Your body is like a car. Over time, things start to wear out — parts get rusty, the engine runs slower, and things don't work as well as they used to. That's kind of what happens when we get older.

Klotho is like a super mechanic that lives inside your body. It goes around fixing things, cleaning up rust, and keeping your engine running smoothly. People who have lots of Klotho stay healthier longer — their brains think better, their hearts pump stronger, and they feel more energetic.

The cool part? Scientists are figuring out how to give people more Klotho, which could help everyone stay healthier as they grow up and get older!

2
High School
Ages 14-18

Historical context: Klotho was discovered in 1997 by Dr. Makoto Kuro-o at the University of Texas. He found it by accident while studying mice with a genetic mutation. These mice aged incredibly fast — they developed osteoporosis, hardened arteries, and died young. When he traced the cause, he found a single defective gene. He named the protein it produces "Klotho" after the Greek Fate who spins the thread of life.

The discovery was revolutionary because it suggested aging might be controlled by specific genes rather than being inevitable decay. If one broken gene could accelerate aging so dramatically, maybe fixing or boosting that gene could slow it down.

What Klotho does:

  • Anti-aging hormone: Circulates in your blood and protects organs throughout your body
  • Brain protector: Enhances cognition and protects against neurodegenerative diseases
  • Kidney guardian: Primarily produced in kidneys; kidney disease causes Klotho to plummet
  • Inflammation fighter: Suppresses chronic inflammation, a key driver of aging

The aging connection: Klotho levels naturally decline as we age — a 70-year-old has about half the Klotho of a 20-year-old. This decline correlates with age-related diseases: heart disease, dementia, osteoporosis, and cancer.

The Human Evidence

A genetic variant called KL-VS gives some people naturally higher Klotho levels. Studies show these people live longer, score better on cognitive tests even into old age, and have larger brain regions associated with memory. About 20% of people carry this "longevity variant."

3
College Undergraduate
Ages 18-22

Molecular biology of Klotho:

The KL gene encodes a single-pass transmembrane protein with two forms:

  • Membrane-bound Klotho: Functions as a co-receptor for FGF23 (fibroblast growth factor 23), regulating phosphate and vitamin D metabolism
  • Soluble Klotho (sKlotho): The cleaved ectodomain that circulates in blood, CSF, and urine — this is the "longevity hormone" form

Key signaling pathways:

  • FGF23-Klotho axis: Regulates phosphate homeostasis. Klotho is required for FGF23 to signal properly; without it, phosphate accumulates and causes vascular calcification
  • Wnt signaling inhibition: Klotho suppresses Wnt, which when overactive drives cellular senescence and fibrosis
  • Insulin/IGF-1 pathway: Klotho modulates insulin signaling, connecting it to the well-established longevity pathway
  • Oxidative stress reduction: Upregulates antioxidant enzymes (SOD, catalase) and downregulates NADPH oxidase

Tissue expression and effects:

  • Kidney: Primary source; Klotho decline precedes and accelerates chronic kidney disease
  • Brain: Expressed in choroid plexus; protects neurons, enhances synaptic plasticity, boosts NMDA receptor function
  • Heart: Protects against cardiac hypertrophy and fibrosis
  • Vasculature: Prevents endothelial dysfunction and calcification
The Cognitive Connection

In 2014, Dena Dubal at UCSF showed that elevating Klotho in mice improved their performance in cognitive tests — regardless of age. The mice showed enhanced long-term potentiation (LTP), the cellular basis of learning. This sparked intense interest in Klotho as a cognitive enhancer, not just a longevity factor.

4
Graduate Student
Advanced degree level

Klotho's mechanism in cognition:

The cognitive benefits of Klotho appear to work through GluN2B-containing NMDA receptors. Klotho enhances GluN2B surface expression and synaptic localization, increasing NMDA receptor function in the hippocampus and prefrontal cortex. This explains the enhanced LTP and improved spatial and working memory observed in Klotho-overexpressing mice.

Crucially, blocking GluN2B abolishes Klotho's cognitive benefits, confirming the mechanism. This suggests potential therapeutic strategies: either boost Klotho directly, or target the downstream GluN2B pathway.

Klotho and the aging kidney:

Chronic kidney disease (CKD) is essentially a state of Klotho deficiency. As kidney function declines, Klotho production drops, creating a vicious cycle:

↓Klotho → ↑FGF23 → ↑Phosphate → Vascular calcification → ↓Kidney function → ↓Klotho

This explains why CKD patients have accelerated cardiovascular aging (calcified arteries, cardiac hypertrophy) and cognitive decline — they're experiencing systemic Klotho deficiency.

Therapeutic approaches under investigation:

  • Recombinant Klotho protein: Direct administration of soluble Klotho; challenges include short half-life and delivery
  • Klotho gene therapy: AAV-mediated delivery of the KL gene; promising in mice but human translation is years away
  • Klotho-boosting compounds: HDAC inhibitors, vitamin D, and exercise all increase Klotho expression
  • Anti-FGF23 antibodies: Burosumab already approved for XLH; may have broader aging applications
The Parabiosis Experiments

When old mice are surgically connected to young mice (sharing blood circulation), the old mice show rejuvenation. Recent work suggests Klotho is one of the key "young blood factors." Injecting Klotho protein into old mice recapitulates some — but not all — of parabiosis benefits, particularly in the brain.

5
Expert
Researchers & practitioners

Klotho structure-function and fragment biology:

Soluble Klotho exists in multiple forms: the full-length ectodomain (~130 kDa) and two KL domains (KL1 and KL2) that can be independently cleaved. Recent structural work reveals that KL1 retains most anti-aging activity, including FGF23 co-receptor function and Wnt inhibition. KL2's role remains less clear, though it may have independent sialidase activity affecting glycoprotein function.

ADAM10 and ADAM17 (α-secretases) cleave membrane Klotho to generate sKlotho. This cleavage is regulated by inflammatory cytokines and may be a therapeutic target — enhancing α-secretase activity could increase circulating Klotho.

Klotho in cancer — a paradox:

Klotho generally acts as a tumor suppressor: it inhibits Wnt/β-catenin (oncogenic in many cancers), IGF-1 signaling, and TGF-β pathway. Klotho is epigenetically silenced in multiple cancers (breast, lung, colon, liver) and its re-expression suppresses tumor growth.

However, the relationship is complex. FGF23-Klotho signaling can promote certain tumors, and very high Klotho might enhance cancer cell survival under stress. The therapeutic window needs careful definition.

Biomarker and clinical translation challenges:

  • Assay variability: Commercial sKlotho ELISAs show poor correlation; fragment-specific assays needed
  • Reference ranges: No established normal ranges; values vary 300-1000 pg/mL depending on assay
  • Tissue vs. circulating: sKlotho may not reflect tissue Klotho levels; brain Klotho particularly difficult to assess
  • Intervention trials: No completed human trials of Klotho enhancement; CKD population likely first indication

Emerging directions:

  • Klotho mimetics: Small molecules that mimic Klotho's Wnt-inhibiting function without full protein complexity
  • CNS delivery: Klotho's large size limits BBB penetration; nanoparticle and focused ultrasound delivery being explored
  • Combination with senolytics: Klotho + senolytic drugs may synergize for anti-aging effects
The 2023 Breakthrough

Dubal's group showed that a single injection of Klotho protein fragment (KL1) improved cognitive function in both young and old mice within hours — and the effect lasted weeks. The fragment doesn't cross the BBB, suggesting peripheral Klotho triggers central effects, possibly via the choroid plexus or vagus nerve. This "peripheral-to-central" mechanism opens new therapeutic possibilities.

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